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大連雅思考試試題
無(wú)論是在學(xué)校還是在社會(huì)中，我們總免不了要接觸或使用試題，試題可以幫助主辦方了解考生某方面的知識(shí)或技能狀況。你知道什么樣的試題才是好試題嗎？以下是小編整理的大連雅思考試試題，僅供參考，希望能夠幫助到大家。
L--V67
R--V66
W--V115
聽(tīng)力V67回憶
section 1 印度女生的accommodation form second name(Bhatt)，生日(31 March 1972)，住幾年(2)，住宿類型(bedside)，愛(ài)好，習(xí)慣(no red meat)，和誰(shuí)住(student)，有什么要求(有shared room)
section 2 picture和movie
7——8道選擇題3選1，第一道是圖中馬的幾條腿離地？
還有誰(shuí)問(wèn)發(fā)明的什么玩意兒？
然后3-4個(gè)填空問(wèn)有一部電影是什么？
還有一部電影地意思用了什么填聲音什么的？
還有一個(gè)電影第一次用了subtitle因?yàn)?sound bad)
section3 是一個(gè)表格，好像是教授問(wèn)兩個(gè)學(xué)生一男一女，
答案上下左右次序挺亂，有愛(ài)好什么的，
section4 先是填空將學(xué)校的設(shè)施在什么位置等，
然后是地圖，讓你在7-8各選項(xiàng)中選出4個(gè)填在空著名稱的建筑上。
Reading
第一篇就是V25的第一篇！關(guān)于rhythm。
閱讀第二篇講阿拉斯加捕魚業(yè)
第三篇講語(yǔ)言變化，有可能是V27的SECTION3
都比第一篇簡(jiǎn)單些。
writing V115
Task 1
關(guān)于2000年，英國(guó)的犯罪率，兩個(gè)圖，第一個(gè)根據(jù)年齡的曲線圖，第二個(gè)是pie chart.是關(guān)于囚犯性別的，女4%，男96%。
Task 2
有人認(rèn)為不應(yīng)該殺動(dòng)物，因?yàn)槟鞘菤埲毯筒槐匾模腥苏J(rèn)為肉類是必要的，談一下這兩方面和你的觀點(diǎn)。
Some people think killing animals for food is cruel and unnecessary and some people think is essential for dairy diet ...To extend what do you think about the both issues?
試題二
1. The failure of a high-profile cholesterol drug has thrown a spotlight on the complicated machinery that regulates cholesterol levels. But many researchers remain confident that drugs to boost levels of ’good’ cholesterol are still one of the most promising means to combat spiralling heart disease.
2. Drug company Pfizer announced on 2 December that it was cancelling all clinical trials of torcetrapib， a drug designed to raise heart-protective high-density lipoproteins (HDLs)。 In a trial of 15000 patients， a safety board found that more people died or suffered cardiovascular problems after taking the drug plus a cholesterol-lowering statin than those in a control group who took the statin alone.
3. The news came as a kick in the teeth to many cardiologists because earlier tests in animals and people suggested it would lower rates of cardiovascular disease. There have been no red flags to my knowledge， says John Chapman， a specialist in lipoproteins and atherosclerosis at the National Institute for Health and Medical Research (INSERM) in Paris who has also studied torcetrapib. This cancellation came as a complete shock.
4. Torcetrapib is one of the most advanced of a new breed of drugs designed to raise levels of HDLs， which ferry cholesterol out of artery-clogging plaques to the liver for removal from the body. Specifically， torcetrapib blocks a protein called cholesterol ester transfer protein (CETP)， which normally transfers the cholesterol from high-density lipoproteins to low density， plaque-promoting ones. Statins， in contrast， mainly work by lowering the ’bad’ low-density lipoproteins.
Under pressure
5. Researchers are now trying to work out why and how the drug backfired， something that will not become clear until the clinical details are released by Pfizer. One hint lies in evidence from earlier trials that it slightly raises blood pressure in some patients. It was thought that this mild problem would be offset by the heart benefits of the drug. But it is possible that it actually proved fatal in some patients who already suffered high blood pressure. If blood pressure is the explanation， it would actually be good news for drug developers because it suggests that the problems are specific to this compound. Other prototype drugs that are being developed to block CETP work in a slightly different way and might not suffer the same downfall.
6. But it is also possible that the whole idea of blocking CETP is flawed， says Moti Kashyap， who directs atherosclerosis research at the VA Medical Center in Long Beach， California. When HDLs excrete cholesterol in the liver， they actually rely on LDLs for part of this process. So inhibiting CETP， which prevents the transfer of cholesterol from HDL to LDL， might actually cause an abnormal and irreversible accumulation of cholesterol in the body. You’re blocking a physiologic mechanism to eliminate cholesterol and effectively constipating the pathway， says Kashyap.
Going up
7. Most researchers remain confident that elevating high density lipoproteins levels by one means or another is one of the best routes for helping heart disease patients. But HDLs are complex and not entirely understood. One approved drug， called niacin， is known to both raise HDL and reduce cardiovascular risk but also causes an unpleasant sensation of heat and tingling. Researchers are exploring whether they can bypass this side effect and whether niacin can lower disease risk more than statins alone. Scientists are also working on several other means to bump up high-density lipoproteins by， for example， introducing synthetic HDLs. The only thing we know is dead in the water is torcetrapib， not the whole idea of raising HDL， says Michael Miller， director of preventive cardiology at the University of Maryland Medical Center， Baltimore.
(613 words nature)
Questions 1-7
This passage has 7 paragraphs 1-7.
Choose the correct heading for each paragraph from the list of headings below.
Write the correct number i-ix in boxes 1-7 on your answer sheet.
List of Headings
i. How does torcetrapib work?
ii. Contradictory result prior to the current trial
iii. One failure may possibly bring about future success
iv. The failure doesn’t lead to total loss of confidence
v. It is the right route to follow
vi. Why it’s stopped
vii. They may combine and theoretically produce ideal result
viii. What’s wrong with the drug
ix. It might be wrong at the first place
Example answer
Paragraph 1 iv
1. Paragraph 2
2. Paragraph 3
3. Paragraph 4
4. Paragraph 5
5. Paragraph 6
6. Paragraph 7
Questions 7-13
Match torcetrapib，HDLs，statin and CETP with their functions (Questions 8-13)
Write the correct letter A， B， C or D in boxes 8-13 on your answer sheet.
NB You may use any letter more than once.
7.It has been administered to over 10，000 subjects in a clinical trial.
8.It could help rid human body of cholesterol.
9.Researchers are yet to find more about it.
10. It was used to reduce the level of cholesterol.
11. According to Kashyap， it might lead to unwanted result if it’s blocked.
12. It produced contradictory results in different trials.
13. It could inhibit LDLs.
List of choices
A. Torcetrapic
B. HDLS
C. Statin
D. CETP
(by Zhou Hong)
Suggested Answers and Explanations
1. vi
2. ii
3. vii 本段介紹了torcetrapib和statin的治病原理，但是同時(shí)短語(yǔ)in contrast與之前第二段后半段的內(nèi)容呼應(yīng)，暗示了這兩種藥在理論上能相輔相成，是理想的搭配。第一個(gè)選項(xiàng)無(wú)法涵蓋整段意義，故選擇i是錯(cuò)誤的。
4. iii 本段分析了可能導(dǎo)致torcetrapibl臨床試驗(yàn)失敗的原因，后半段指出如果以上推測(cè)正確，那么未來(lái)的藥物可借鑒這個(gè)試驗(yàn)，設(shè)法避免torcetrapib的缺陷，研制出有效的藥物。viii選項(xiàng)無(wú)法涵蓋后半段的意思。
5. ix 見(jiàn)首句。
6. v
7. A 見(jiàn)第二段。題目中administer一詞意為用藥，subject一詞為實(shí)驗(yàn)對(duì)象之意。
8. B 見(jiàn)第四段… to raise levels of HDLs， which ferry cholesterol out of artery- clogging plaques to the liver for removal from the body.即HDLs的作用最終是將 choleserol清除出人體：… for removal from the body。
9. B 見(jiàn)第四段But HDLs are complex and not entirely understood.
10. C 見(jiàn)第二段… plus a cholesterol-lowering statin，即statin是可以降低cholesterol的。
11. D 見(jiàn)第六段 So inhibiting CETP， … might actually cause an abnormal and irreversible accumulation of cholesterol in the body.
12. A 見(jiàn)第三段。
13. C 見(jiàn)第四段Statins， in contrast， mainly work by lowering the ’bad’ low-density lipoproteins.
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